Amazing threads like these still get started to this day, knowing the amount of research and studies that have surfaced in the past 10 years. Along with numerous lyme patients reaching remission with a new drug named disulfiram..
dcd2103 said...
I bought some college text books on immunology, and have been reading about how the immune system works for several months. After studying it I was pretty positive that there were several parts of the immune system that were going haywire at the same time. Lyme can cause cytokine lead autoinflammatory responses through the innate and cell mediated branches of the immune system which leads to symptoms like fatigue and joint pain, and it can also cause humoral branch B cell/anti body type autoimmunity, which is why some of us develop things like small fiber neuropathy.
First off, none of the college books you're reading will ever cover babesia and bartonella, because new studies are just surfacing about
how resilient these these pathogens are, these two are still in very unknown territory and not very well studied. I'm not sure how you could be convinced an old book about
immunology would cover both babesia and bartonella... You need to be reading new studies, not old...
https://news.yale.edu/2018/11/29/yale-scientists-develop-new-system-study-emerging-tickborne-diseaseYale.edu said...
They also tested four current drugs that are used to treat the disease and found that the parasite has low susceptibility to these therapies.
https://www.northcarolinahealthnews.org/2013/12/05/bartonella-is-everywhere-so-why-dont-we-know-more-about-it/Dr. Ed Breitschwerdt who has studied Bartonella for 30 years said...
You cannot float humans or horses in enough Doxycycline to kill this bacteria
So you're basically taking two already resistant pathogens, combining them with another bacteria similar to syphilis which can cross the blood brain barrier, and even manifest in bone.
https://youtu.be/44xl0z8i5x8?t=29I think you also need to read up on Dr. Sapi's new study that was recently published, showing biofilms and spirochetes in a patient after 16 years of antibiotics.
https://www.mdpi.com/2079-6382/8/4/183/htmdcd2103 said...
So this brought me to the idea that all of these antibiotics and disulfirams and ozones and treatment options aimed at KILLING the lyme are pointless.
If you fully understood autoimmunity, you'd understand that an imbalanced microbiome is a major connection to autoimmunity. And Dr. Kim Lewis's point he makes with disulfiram is, it doesn't kill off any of the natural cultures in our gut. So you're thought of disulfiram just KILLING is a total misconception! This is one of the major benefits of the drug, unlike chemo or antibiotics in general, it targets the pathgones without harming your microbiome which is very important for keeping a healthy immune system and not causing autoimmunity.
Be aware, Dr. Rajadas from Stanford already said disulfiram may win the molecule of the year award due to it's multi-use, not just for lyme disease, but AIDs and even Cancer. And another reason this drug isn't all about
killing, is it's been found to wake up the dormant cells in the AIDs virus, making them vulnerable to antivirals.
And really, you need to be reading up on each specific pathogen, rather than reading old books about
autoimmunology and think it's going to cover all these tick pathogens and diseases. That's a very archaic way to think, that immunity is going to explain and cover them all. It's just like how a lot of Doctors think after they graduate from college and think just because they have a Phd and have studied for 8 years, that there's nothing new to learn. Science is changing on a day to day basis and as we progress with technology, it will soon be on an hourly basis. I suspect when we hit the robot self aware point, who knows how quick breakthroughs will be made.
See you're arguing in a predate time manner, using old school thought and studies, not even considering any of the new studies. And already you seem to have a predisposition bias considering you think disulfiram is an antibiotic. It's not even an antibiotic, never was. I expect this thread to go nowhere just like any of the arguments from the IDSA or CDC side ever does.
You do realize that 28 lyme patients are suing the IDSA Doctors for conspiring 8 Insurance Companies and so far one insurance company has already settled?
https://www.courthousenews.com/insurers-accused-conspiring-deny-lyme-disease-coverage/You do realize that the borrelia bacteria has a history of being biowarfare and the whistleblower who came forward is the person who it's named after?
https://youtu.be/nrpjwtdzcjq?t=560Anyways, I'm not looking to turn your ideology nor argue with you, as you obviously have a predisposition bias, everything I have wrote is already on the web, and if you knew any of it, you'd consider at least one of these studies as factual evidence, but you haven't, you're very one sided. And if you look what I've said, I've suggested how Lyme Disease can be both a chronic infection and autoimmune at the same time, so the question should be why can't Lyme actually be both.
If you've noticed, a disease like lyme and chronic infection isn't anything new. If you look at what's happening with Tuberculosis over the years, Leprosy, as well as other infections like MRSA. These bacteria are evolving, becoming more resistant to antibiotics each and every year, it's called EVOLUTION. The one thing that keeps these infections is these itty bitty things called persister cells, maybe you should do a little modern day reading on persister cells, rather than old books on immunity.
Evolution of bacteria, drug resistance, persister cells, and biofilms should be in your vocabulary, but sorry to say, I don't see one single mention of any of these...
Post Edited (Charlie55) : 12/12/2019 1:22:21 AM (GMT-7)