Posted 10/16/2015 9:58 PM (GMT 0)
Hi I apologize in advance for a lengthy post. I'm new to the forum but not uc. Report info is below.
I was diagnosed with uc about 20 years ago can't remember date. I was on Asacol for about 10 years then I stopped ago med and was able to keep myself in control if a flare was starting I would change my diet and ease up on what I was doing and sometimes take some of my Dad's prednisone but I avoid prednisone until I know I can't go without something. I hate going to Dr about this so that's my problem also. I had a dr who if I walked into his office without an appointment and he hasn't seen me in a long time he wound tell his nurse to get me in a room right away. But he moved away. :( Fast forward to about May this year and I started a flare but it would ebb and flo bleeding come and go so I was handling it until about August when boom at least 20 times a day blood, mucus and pain but I was traveling so I waited to till I got home and couldn't get into Dr for 3 weeks when I finally realized I needed help now so ER I went they did test and ctscan told me ctscan results would take 2 to 2 1/2 hours I got wheeled back to my room went to use restroom and Dr s there waiting for me telling me I needed to be admitted. Now I don't understand these results. Especially the CT and the scope say active uc and the biopsy say quiescent IBD could it go into remission in 2 weeks and also scope looked active and still having about 5 to 10 bm a day. Do I have uc? Again I apologize for the long post but appreciate any help. Meds right now Asacol 800 mg, budesonide 9 mg
Ctscan report
Small benign hemangioma in the left lobe of the liver. Liver is
otherwise unremarkable. Status post cholecystectomy with normal
prominence of the biliary tree. The pancreas, spleen, adrenal glands,
and kidneys are negative.
The distal esophagus, stomach, and small bowel are normal. The
appendix is normal. There is diffuse colonic wall thickening from
cecum through the rectum with prominent pericolonic inflammatory
changes. There is no evidence of associated perforation; no free fluid
or free air.
The abdominal vasculature including the aorta and IVC is negative. No
grossly pathologic abdominal adenopathy however there are multiple
sub-5 mm scattered mesenteric lymph nodes present in the colonic
mesentery.
The bladder is partially filled and grossly normal. Ovaries are unremarkable.
Bones are negative.
IMPRESSION:
Pan colitis. The leading considerations are inflammatory bowel disease
such as ulcerative colitis or Crohn's versus an infectious colitis,
most commonly C. difficile. No evidence of complication by perforation
or abscess.
Colonoscopy report
Findings:
MILD ULCERATIVE COLITIS LEFT INVOLVED MORE THAN RIGHT COLON.
The perianal and digital rectal examinations were normal.
Inflammation characterized by erythema, friability, granularity,
scarring, aphthous ulcerations, serpentine ulcerations and shallow
ulcerations was found as medium-sized patches surrounded by normal
mucosa in the rectum, as medium-sized patches surrounded by normal
mucosa in the recto-sigmoid colon, as medium-sized patches surrounded by
normal mucosa in the sigmoid colon and as medium-sized patches
surrounded by normal mucosa in the descending colon. This was mild in
severity. Biopsies were taken with a cold forceps for histolog
y. FROM RT
COLON AND LT COLON.,
Impression: - Inflammation was found in the rectum, in the
recto-sigmoid colon, in the sigmoid colon and in the
descending colon secondary to ulcerative colitis.
Biopsied.
Recommendation: - Use Asacol 800 mg PO TID.
- Use budesonide 9 mg PO one time per day.
- Follow up with me in 2 months.
Biopsy report
Final Pathologic Diagnosis
1. Random right colon biopsies: Moderate to marked crypt distortion with
no evidence of active colitis, compatible with quiescent inflammatory bowel
disease.
2. Random left colon biopsies: Marked crypt distortion with no evidence of
active colitis, compatible with quiescent inflammatory bowel disease.
3. Duodenum, biopsies: Intact villiform duodenal mucosa -- there is no
evidence of villous blunting, intraepithelial lymphocytosis or crypt
hyperplasia.