Dr. says fibro causes my ringing and hearing loss

I finally get in with a dr. (ent) to discuss ringing in ears, temporary loss of hearing, and mild to severe vertigo (always present).

He says that I have a broken bone in my ear (omg, there is bone in ear??!!!)

and that I should have hearing loss, but I dont. YAY!! so now the doctor scratches his head because my hearing is normal (that was priceless)

He says that the ringing and short-term hearing loss is due to the tension in the muscles in my ears and that it is common with fibro. So he is recmding to my pcp, to be put on muscle relaxers.

He says that it is not meniere.s and no ent reason for vertigo

My rheumy said that my thyroid was off, but the ent says that it is the "stimulator" that is high, and that the actual thyroid output is normal. That it would only be a problem if there was extra stimulator, yet not enough thyroid output. He was a sweet dr (all smiles) until I mentioned t3 and t4.   He did one of these   then explained about the stimulator.

I am a little frustrated about going in circles with doctors and that the ent was the only dr I have seen that has talked to me about how fibro is affecting my body.

i fought to see a rhuemy at a major university and she is head of the department..... and the ent is the only one who is relating fibro to me in personal way.

like I said, he was nice. He is even going to remove an oral mass for me on wed. Something else that I wasnt able to get my Dr. to go ahead and deal with. It is the size of...from the tip of my pinky to the second knuckle. On the inside of my cheeck. makes it hard to chew and talk. I think it was caused by braces as a teenager. Why I shared that I dont know, just excited I guess.

 

Just one more thing. Vertigo with hearing loss that fluctuates along with tinnitus sounds like Meniere's.
With Meniere's, it's believed that food allergy plays a part.
I've noticed my tinnitus reaches screaming levels when my nervous system is reacting to other issues -- can be food related or medication related or sleep deprivation.

I would definitely check your meds if you are on any. Almost everything affects my ears.
Also, are you gluten free? Yeast free? Dairy free? Any other food allergies or sensitivities?

The gastrointestinal system is closely connected to the brain from whence the tinnitus originates.
In fact the gastro tract is known as the second brain and has neurotransmitters in abundance.

If I could ever fix my gut, I think I might fix a whole lot of other issues ...

Just some food for thought.

yeah, thanks AB. I was floored and he is just some ent. A dr saying he can feel the fibro tension--was like a gol dmedal. Yay, they see it!!!!

and thankyou Marrit, for the interesting post. Have you found a dr to help with the mitochondrial dysfunction. I wouldnt know where to look. It seems that they dislike the mention of it even worse than fibro. I saw your other post about it and will be looking into this alot more. Thanks for this interesting lead.

Yes, I have the skin sensations and pain, also have psoriasis.

And if they could fix my gut, I too would probably fix alot of other issues.

SimplyUs and anyone else with painful skin issues ("Substance P"), here is a medical research abstract I found relating to mast cell activation:


Acute Immobilization Stress Triggers Skin Mast Cell Degranulation via Corticotropin Releasing Hormone, Neurotensin, and Substance P: A Link to Neurogenic Skin Disorders

Leena K. Singh,
Xinzhu Pang,
Nicholas Alexacos,
Richard Letourneau,
Theoharis C. Theoharides

Department of Pharmacology and Experimental Therapeutics, Tufts University School of Medicine, Boston, Massachusetts, 02111

http://dx.doi.org/10.1006/brbi.1998.0541, How to Cite or Link Using DOI


Abstract

Many skin disorders, such as atopic dermatitis and psoriasis, worsen during stress and are associated with increased numbers and activation of mast cells which release vasoactive, nociceptive, and proinflammatory mediators. Nontraumatic acute psychological stress by immobilization has been shown to induce mast cell degranulation in the rat dura and colon. Moreover, intradermal injection of corticotropin-releasing hormone (CRH) or its analogue urocortin (10−5–10−7 M) induced skin mast cell degranulation and increased vascular permeability. Here, we investigated the effect of acute immobilization stress on skin mast cell degranulation by light microscopy and electron microscopy. Immobilization for 30 min resulted (P < 0.05) in degranulation of 40.7 ± 9.1% of skin mast cells compared to 22.2 ± 7.3% in controls killed by CO2 or 17.8 ± 2.4% in controls killed by pentobarbital. Pretreatment intraperitoneally (ip) with antiserum to CRH for 60 min prior to stress reduced (P < 0.05) skin mast cell degranulation to 21.0 ± 3.3%. Pretreatment with the neurotensin (NT) receptor antagonist SR48692 reduced (P < 0.05) mast cell degranulation to 12.5 ± 3.4%, which was significantly (P < 0.05) below control levels. In animals treated neonatally with capsaicin to deplete their sensory neurons of their neuropeptides, such as substance P (SP), mast cell degranulation due to immobilization stress was reduced to about 15%. This is the first time that stress has been shown to trigger skin mast cell degranulation, an action not only dependent on CRH, but apparently also involving NT and SP. These findings may have implications for the pathophysiology and possible therapy of neuroinflammatory skin disorders such as atopic dermatitis, neurogenic pruritus, or psoriasis, which are induced or exacerbated by stress.

http://www.sciencedirect.com/science/article/pii/S0889159198905414