The 7 Provocative Findings of the Dr. P. Duray Research Fellowship Foundation …and what they mean

Full title of the artcle:
"The Seven Provocative Findings of the Dr. Paul Duray Research Fellowship Foundation
…and what they mean to Medicine and Patients."

by Tom Grier , Microbiologist

/durayresearch.wordpress.com/about-2/7-provocative-findings-intro/

Please note: a more accessible version of this article for patients with neuroborreliosis can be downloaded at
/durayresearch.files.wordpress.com/2016/07/7-discoveries-pdf-final-july16-sansmkup.pdf

This article is PACKED with information, so I will only share a small amount - to hopefully wet your appetite for more!!

The 7 'parts':

Mother-to-child transmission of Borrelia across the womb
Finding Borrelia burgdorferi and miyamotoi associated with Amyloid Plaques in Alzheimer’s disease brains
Finding Borrelia in Lewy Body Dementia
Nematode worms found in the CSF (spinal fluid) of Multiple Sclerosis patients
Nematode worms found in Alzheimer’s brains
Borrelia found in five deadly brain tumors (Glioblastoma multiforme)
Borrelia Mayonii and Borrelia burgdorferi found in human testicle

"Since 1975 when Lyme disease was first introduced to the medical literature, it has been surrounded by controversy and misunderstandings. Much of the problem stemmed from trying to understand this disease entirely through antibody tests (serology) based entirely on just one species – Borrelia burgdorferi.



We now know that there are many species of Lyme disease. Borreliosis is not just one disease, it is part of a family of diseases that can no longer be considered separate or isolated from Lyme disease. The best example of this is Borrelia miyamotoi. It is found in hard-shelled ticks just like Lyme disease, but it is a Relapsing Fever borrelia. It took over 10 years for microbiologists to place it in the Relapsing Fever category as opposed to the Lyme-genetic grouping.



Not surprising is the fact that B. miyamotoi is found in the human brain right alongside B. burgdorferi. They may look like two separate diseases on paper, but in the human brain they are pathogens and must be eradicated together.



An even more concerning part of the Lyme disease story is that virtually no funding in any country has been put into Borrelia pathology. The fact is Lyme serology simply cannot detect this family of bacteria with any reliability, mostly because Borrelia, once inside the human brain, is behind the blood-brain-barrier, and inside human brain cells. As a result, the human immune system can no longer recognize it.



Add to this the fact there are more than a dozen species of Borrelia that cause Lyme disease, many of which can penetrate any tissue, and add a couple Relapsing Fevers that tag along for the ride, and it becomes clear that the Lyme disease blood tests based on Borrelia burgdorferi detection that have been used for 30 + years have become pretty much useless."

Excellent stuff... the info keeps coming...

-p

Hi Traveler!-

Thank you for sharing this!!!! :)

It just blows my mind that this valuable information NEVER gets the Attention that it deserves!

This information should be in every medical book, reported through the media, and of course most of all ACKNOWLEDGED by the A-holes that call themselves the CDC!

Oh, but wait, if we could cure some of these diseases , then big pharma wouldn't be able to keep emptying so many people's wallets.

It doesn't need to make sense, it needs to make dollars!!! $$$$$

Grrrrrrrrrrrrrrrrrrr!

-----------------------------------------
1985 NIH - DURAY with AC STEERE

Lyme arthritis. Spirochetes found in synovial microangiopathic lesions.
Y. E. Johnston, P. H. Duray, A. C. Steere, M. Kashgarian, J. Buza, S. E. Malawista, and P. W. Askenase

www.ncbi.nlm.nih.gov/pmc/articles/PMC1887850/
-----------------------------------------------------
1986 AB MACDONALD

"Human fetal borreliosis, toxemia of pregnancy, and fetal death"

www.ncbi.nlm.nih.gov/pubmed/?term=3554838
This one spells it out more:
www.sciencedirect.com/science/article/pii/S0176672486801225/part/first-page-pdf
-----------------------------------------------

"Stillbirth following maternal Lyme disease"
MacDonald AB, Benach JL, Burgdorfer W.
N Y State J Med. 1987 Nov;87(11):615-6.
PMID: 3480464
----------------------------------------------------
1992
"The NIH-3 immunodeficient mouse is a model for Lyme borreliosis myositis and carditis"
D. L. Defosse, P. H. Duray, and R. C. Johnson www.ncbi.nlm.nih.gov/pmc/articles/PMC1886584/
------------------------------------------------------
1992
S. Czub, P. H. Duray, R. E. Thomas, and T. G. Schwan

Cystitis induced by infection with the Lyme disease spirochete, Borrelia burgdorferi, in mice.

www.ncbi.nlm.nih.gov/pmc/articles/PMC1886652/
-------------------------------------------------
Many others, many authors, from many geo areas...

Lyme borreliosis in Poland in 2009
In 2009, 10 329 cases of Lyme borreliosis were reported in Poland, more about 20% than in the last year and more about 25% higher incidence--27.1 per 100 000, and above two times more than the median incidence in 2003-2007. The highest incidence 114.0 per 100 000 was registered in podlaski voivodeship. Of total Lyme borreliosis cases 2 504 were hospitalized. PMID: 21913475

I agree! And the amount of progress they are now making is really quite remarkable, and so needed!

There is literally an avalanche of research going on, but the right people continue to do everything they can do to discredit and ignore it.

Elena! I knew we would hear from you! As always, I'm very, very happy to share important and very excellent articles from knowledgeable and trustworthy sources!!

Thank you for what you do too!!

There are a number of worms that have been known for a long time to be able to cause terrible braininfections.

However this is the first time that a researcher has found worms in the brain with Borrelia sheltering inside them.

The worms will provide the perect defense against our immune system and against many anti-bacterial medicines.

Angiostrongylus cantonensis is a Souteast Asian worm which regularly causes meningitis. It is known to damage the optic nerve and can cause blindness and demyelination - just as in MS.

We don't yet know which worms are involved. But Dr Alan MacDonald has found Borrelia and worms in the CSF of patients with MS - as Tom Grier explains in this article.

I don't know if there is a doctor in the house at the moment. I'm a former hospital staff nurse, if that's any help....

But at the end of Tom Grier's article you'll find links to Dr MacDonald's videos which are all publicly available free of charge on Youtube and Vimeo etc - these are well worth watching and you will learn a lot!

Best
Elena

Traveler, thanks for posting this here and on our Facebook page. I centered discussions at our group meeting this week on some of what you had in this article. You are a blessing!!

"Lyme Research Alliance (LRA) and Tick-Borne Disease Alliance (TBDA) merged to become Global Lyme Alliance. LRA awards grants where the most promising cutting-edge science is being deployed and this most recent round will support work at Columbia, Johns Hopkins, Northeastern, Rutgers, UCSF, UC Davis, and the University of Pennsylvania."

Sounds nice, doesn't it?

With Luc Montagnier & Judith Miklossy, you would think it would help level the playing field. But then you investigate to find out just HOW far reaching into the educational systems, clinical studies, BayAreaLyme/CDC /Johns Hopkins & all the funding & govt grants & follow the $ & follow the power.

The last study done endorsed by LRA, NIH Mark J. Soloski, with had ALL RHEUMY docs (who, btw, have a vested interest in perpetuating their livelihood), to conduct their "clinical study" stated that anyone with "history of prior Lyme disease or the presence of confounding preexisting medical conditions associated with prolonged fatigue, pain or neurocognitive symptoms" need not apply. Well, what a wonderful carousel ride! (Here's that LRA/TBDA/NIH/govt funded "study" link:
http://journals.plos.org/plosone/article?id=10.1371%2Fjournal.pone.0093243)

I would LOVE to BELIEVE that good will triumph over evil, but the biolabs continue without any oversight, the media-fo'gettaboutit---& the public zzzz. But sure, yeah, spread the word.

I think that Happyjo is correct to flag up the fact that a lot of our most wealthy Lyme funding groups are getting into bed with the wrong people and funding the wrong researchers.

At this moment in time, virtually ANY Lyme study that receives NIH or CDC funding or praise is highly likely to be bad science. We can't ignore the fact that there is a deliberate obfuscation of the science at the federal government level due to Borrelia's status as an organism of intense interest to biowarfare scientists.

However, I feel that Traveler is right too -we must not give in!

The Lyme charities who are blindly and ignorantly pouring funds into people who are clearly NOT on our side could be called to account by the Lyme community.

We need to question them, ask them what they are doing, and why. Too many people give money and then don't take the trouble to find out how their donations are being spent.

In the past Lyme charities have been involved with funding the likes of IDSA guidelines author Linda Bockenstedt, whose appalling and falsified study purported to show that biofilms of Borrelia were just dead "debris".

Debris of dead bacteria does not normally persist in the human body. It is swallowed and disposed of by immune cells (macrophages). When Dr MacDonald requested that she share some of the "debris" with him so he could test it with a stain that differentiates between dead and live material, she refused.

The Bay Area Lyme Foundation has a section on its website called "Our Scientists". Among them is Alan Barbour (!)

Epidemic Intelligence Service officer and biowarfare scientist Alan Barbour is one of the worst Denialists in history. He has written entire books aiming to prove that chronic Lyme does not exist. How on earth is he one of "our scientists"? He should be listed under a section: "Their Scientists".

All Lyme serology is ultimately based on the strain Barbour gave to the world -Bb sensu stricto B31. But this is a lab strain that has never been found in a human, and its antigens differ signficantly from many of the strains and species that we now know cause terrible Borreliosis infections.

Elena

Good discussion...

It's interesting how different perspectives or exposure shapes our understanding of our world. This is certainly true of Lyme.

I agree with just about everything posted. While nearly all of the scientists "discovering" Bb - the ticks carrying it, and identifying how it manifests into Lyme disease - have a background in biowarfare research (even our beloved Burgdorfer was doing biowarfare research when he found Bb!), Lyme's growth into an endemic problem doesn't just have origins in biowarfare research gone awry.

And it will be decades before any truths of this biowarfare research comes to light. There is no more powerful governing body in this country than the US Military.

But the Lyme endemic is also about pollution, a growing and expanding population and warming climates causing disruption of nature and animal life. Also, its role within an infectious diseases "system" which has huge components within it based entirely on a biowarfare approach to research - the resulting pathogen identification, surveillance and disease definition has been completely engineered. But because of all of these contributing factors, lyme has physically spread into every nook and cranny of the world.

Not because the world is evil, but because it was originally thought to be a small problem in a small, highly affluent part of the world and early on, the science was deeply connected to vaccine pharmaceutical R+D.
- In order to prove the vaccines' efficacy, the definition of Lyme had to remain very narrow.
- So that's why the early science acknowledged only "EM, arthritis, meningitis" versions of Lyme.
- The vaccine trials' "successes" were based on following patients who fell into this category of Lyme and they drew the cutoff during the trials so that the studies showed success, not the full picture of relapse.
- EVERYTHING in the pharmaceutical model--the vaccine science, vaccine-caused harm lawsuit settlements, the business model of the vaccines and other products the pharma co's were going to build from that, the two-tiered serology testing mechanism and process, the CDC surveillance processes, the IDSA Guidelines and continued funding of (specific) scientific research that helps support the early (bad) science... ALL OF IT is interconnected because it's based on the narrow definition of Lyme that underpins this vaccine R+D.

And it's this influence over the science that stunted the development of good science and broad science.

This was allowed because the "Lyme leadership" is the very same small group of researchers who crafted this narrow definition, a group whose notoriety has exploded in the past 10 yrs, this same group maintains control over the Lyme narrative. And that isn't likely going to change any time soon. They have a very tight rein on Lyme leadership and no one else wants to get their hands dirty--not with the CDC (and military in many ways) steering this tanker.

The thing is, their definition of the lyme they've defined isn't necessarily wrong, it's just that it represents 5% AT BEST of what is really going on with Lyme.

There seem to be two mechanisms currently driving research:

1. the autoimmune component of chronic Lyme - while evident in some of the good science is still a narrow definition and certainly does not speak to all chronic manifestations of Lyme. This is the driver for the "treatment centers" which are likely well-funded facilitators of continued research and we can only hope this research leads to successful treatment for these types of Lyme manifestations. You can bet it will lead to continued pharmaceutical investment toward more palliative care drugs because that's where the money is. As mentioned, this is the bread+butter of the Rheumy business model.

2. The next generation of Lyme vaccines, which likely include other tick-borne infections since they involve much simpler science and etiology. The real risk for us here is if these are also based on the original, poorly-defined science. They can't necessarily overturn the bad science with new, good science, or they won't be able to sell anymore of the ogival vaccines (yes, it's still available and they made a European version as well). But they open themselves to more lawsuits. So it will be interesting to see how they got around the bad science and how they're going to develop a vaccine that works.

The other reality is that nearly everyone now has Lyme microbe in their body - different strains, of course. So that's another road block for an effective vaccine, if most of the population is already infected. Too many ways for the vaccine to fail (based on the previous model) so I'm not sure how they're going to get around it. That it's taking so long (they've been talking about it for 15 yrs now - not sure how long they've been in R+D) might also indicate problems with its success.

Unlike other contested diseases, Lyme research has to jump through countless hoops in order to get back on track. But very slowly, components of the Lyme truth are being developed. The current model isn't likely going to change very soon - if at all. The internet is our ally and has had a disastrous impact on the Lyme lies. It's only a matter of time before enough of the right people experience our Lyme experience themselves. While the original "Lyme definition" has continued to challenge any progress from within, significant progress has been made outside of it and this is important.

And this is what I encourage people to focus on. I think it's normal, healthy to go through your own process of inquiry and discovery of why the Lyme controversy developed, why it's such a quagmire in the modern age. But the lie is too big to allow to fail at this point. There are enough smoking guns but the same Sheriff in town.

I was glad to finally understand this information and I've finally compiled a picture of Lyme that is more comprehensive than the sound-bytes that float around our communities, including the very well-done Under Our Skin documentaries (while great, they miss a few pieces of the puzzle). My main objective in this discovery was to be well-informed. But I'll admit I also wanted to know enough to successfully take on the IDSA shills in their socio-political war on Lyme lies marketing campaign and have enjoyed doing so in comment sections on these bogus articles. It's empowering and it's a nice outlet for my frustration.

I still have a little more researching to do, but I'm finally OK leaving some of these truths where they lie and spending energy on discovery that serves us. The critical truth-sharing where it's needed, helping people get the right diagnosis and treatment, and bringing to light the research that IS going in the right direction will eventually reach more people and impact more progress than the current IDSA and CDC Lyme model suffocates.

-p

Speaking of nematodes -
Does anyone know if the product sold for spraying on lawns to kill fleas is, in fact, as safe as claimed? Some accidentally splashed into my eye as I was spraying my lawn for a sudden flea infestation (thanks to an opossum family that decided to squat in my attic).

The product rep assured me it was the kind that "doesn't survive in the human body" but of course I'm paranoid...

-p