Colostrum

Tried years ago, it did nothing.

I also had before, and after labs looking at exactly what you’re looking at ,…and more immune cells too. No improvement.

i tried it recently also

no benefits that i could detect

caused some mild constipation - but nothing too problematic

some of it does - or rather some of the part digested fragments of it that still have antigen neutralising properties make it into the mucosa and potentially the blood stream - whether enough of them do so they have enough effect to be useful is still debateable - probably not

given the normal doses of colostrum are a gram or so - and only a fraction of that is immunoglobulins - and given that humans have about 10g per litre of blood of mainly IgG floating around in them at any one time - i would guess anything making it through the gut lining would be a drop in the ocean systemically.

it is well demonstrated to have positive effects in the gut though - eg in animal farming - eg growth of premature pigs, treating gastric illnesses etc etc

more depth on the topic here
https://www.ncbi.nlm.nih.gov/pmc/articles/pmc4355420/
😀

In the early 1900’s colostrum was thought to be the next anti biotic (which got overshadowed by antibiotics).

There was a US congressman with Lyme who had personalized colostrum made (maybe there’s a more specific name of this?). Essentially a farmer takes blood from in this case the congressman, injects it into the utter of a cow and the colostrum is custom made for the exact things in that blood. This is all on record as he spoke on this in session in congress and claims it brought him into remission.

that is indeed very interesting. it sounds similar to the IgY thing of chickens

I received this information on transfer factor which I thought might be helpful or of interest

Introduction

Transfer factor is a low molecular weight moiety, which is extracted from human leucocytes or bovine colostrum. Its mode of action is the induction of receptors or lymphocytes. It has been used in the therapy of malignancies, autoimmune disease and immune-deficiency disorders. There is evidence that immune complexes mediate the symptoms of food and chemical sensitivities. T suppressor cells are one of the groups of lymphocytes which help in the control of immune complex formation and regulation. Transfer factor enhances suppressor cells, which, in turn, reduce the formation of antigen antibody complexes.

Transfer factor acts as an immune system boost, which is a non-specific immunotherapy, and we have found that many symptoms of patients with multiple sensitivities can be controlled. Often these are people who have had chronic viral infections. We have shown that the T-lymphocyte subsets are returned to normal with the use of vaccines and transfer factor.

We recommend transfer factor for chronic fatigue syndrome or allergic conditions. A blood test, to ascertain subset lymphocytes, should be undertaken first, after which the transfer factor can be prescribed. Extremely sensitive patients may need to be desensitised to the transfer factor, or products from bovine colostrum.

Definition

Transfer factor is an extract from human leukocytes and bovine colostrum which transfers a delayed type of cellular immunity in an antigen-specific manner. Transfer factor also has an antigen-independent effect on cell-mediated immunity and on the inflammatory response, including the prostaglandins, nicotinamide, ascorbic acid, histamine, serotonin, T-lymphocyte maturation, or differentiation factors (thymosin or thymic humoral factors), also cytokines, interleukins and interferons. These components might well produce generalised increases in immunological activity,
and might help in the restoration of a more vigorous immune response by patients.

Historical usage of transfer factor

At the turn of the 20th century, patients with infectious diseases were treated with leukocyte extract by Hiss and his colleagues.1 This was of benefit to patients with infections, but this use was abandoned with the discovery of penicillin and sulfonamides in the 1940s. In 1942, Lansteiner and Chase, 2 showed that delayed hypersensitivity could be transferred in guinea pigs from sensitised donors to non-sensitised recipients using leukocytes, but not serum.

In 1949, Lawrence found that tuberculin and streptococcal sensitivity could also be similarly transferred. He later found that leukocyte extracts could transfer delayed hypersensitivity reactions.3,4 Despite its therapeutic potential, transfer factor was not used until 1970 in the treatment of human disease, 5 when it began to be used for a number of conditions.

Thanks for the Link Loski

it was an interesting read

i don't think i will be rushing out to get my own goat just yet

but may have a look around to see if there is anything more than anecdotal accounts