I learned what Lyme really is and wanted to share - may be upsetting to some!!!

Artemix,

Hi. No I didn't. I even mentioned about that I realize lyme is incurable. That said I was under the impression that many people go into remission and that is what I'm hoping for. I think what got me freaked was the part that if you are feeling good it will come back worse. It's kind of like living with a time bomb. Geesh.

HIV does not own AIDS. AIDS stands for acquired immune deficiency. There are many different types. Like hypocomplementic uticarial vasculitus effects mostly vessels and HIV goes after natural killer cells. There are many, many different types. There are many ways to end up with an acquired immune deficiency, OspA is one way. Mycoplasma can do it. EBV.

I am absolutely bringing this up at my followup. I want that gene test. My LLMD is really open and very knowledgeable and up on all things lyme. He is well known for standing up and advocating for lyme patients and lymes existence. He's one of the good guys.

IHL: Please know that my sharing that I am bringing it up, in no way reflects that I believe this is untrue. I just want to know what my genes reveal and what I'm in for.

Huddie- I am not aware of any test that can determine what type of outcome you will have. My guess is that genes play a role in what outcome you end up with.

The immune panel I mentioned (and what astroman mentioned) only tell you how your immune system is doing. Low numbers mean low function, high numbers mean good function. As far as I know, I'm still pretty new to all these theories myself.

If you have coinfections that are active abx are beneficial. Abx don't treat viruses though. That's where it gets complicated.

IHL - you said you wouldn't do the LDI? Any particular reason why?

I have been hesitant - I don't like not knowing where it's coming from...who is making the 'serum'?
Apparently it all comes from Dr. Vincent. Where does he get it from.

I'm thinking I might try the sublingual one...instead of the injections.


My LLND told me the LDI 'calms down' the immune system...brings it back from the autoimmune state.
So, then it can fight again.

Does that make sense?
I just don't know anymore...

So, what about those of us who don't get well, but don't have high viral loads? (regarding what IHL's Dr. says)

Look I'll give my unvarnished opinion.

This stuff comes from the Kathleen Dickson school on lyme, OspA, the lyme vaccine gave everyone incurable lyme which she tries to classify as a fungal agent. Kathleen makes some decent points, but she goes way off the reservation on too much stuff, she is a divisive antagonistic character in the lyme community. She lords her science background (an undergrad BS) and tries to cudgel people with it into submission. She makes grand sweeping statements to people that these infections are incurable and that no one can get better with antimicrobial treatment. This is a dangerous thing to tell patients who already have little hope. Plus it is straight up wrong. I know countless of stories of people who improved and many more who attained remission because of antimicrobial treatments. In my own case I'd be dead if it weren't for antimicrobial treatments. I get that my story is n=1, but trust Kathleen's work at your own peril.

I don't mean to be unduly harsh. But I am no fan of Kathleen. She tries to couch many of her assumptions as scientific facts and cherry picks the data to fit her narrative. Not saying her work is worthless, I respect some of her stuff, but overall I think she does more harm than good and is more wrong than right. But she is absolutely correct in her analysis on Dearborn.

Post Edited (sebreg) : 9/16/2016 5:23:25 PM (GMT-6)

What is IVIG? And if we already have viral issues (I know I do) that makes healing lyme even harder?

Don't we all have dormant viruses: I read that most of us have EBV, HPV, other herpes type viruses that lay dormant.

Of course we can not kill every bug but we can look at the peer reviewed studies and determine which antibiotic/herbal combo can put Lyme into remission. In acute infection it would be wise to inject Natural killer cells and various other T-cells near the source of the bite. This will clear up the infection and help the immune system inhibit it from spreading. The Lyme bacteria likes to manipulate the immune system and raise IL-10 anti-inflammatory cytokine, so it almost helps to make you feel better for awhile. You'll walk around with it asymptomatically it will reproduce slowly and later lead to a disseminated infection.

Traveler said...

artermix said...
I do not disagree with you Traveler, but since people with white lab coats and 7 years degree framed on walls can make their observations and theories I don't see why we can't either.
Maybe I buy a microscope and do some personal research hands on. I'd love to do that.

That's what this thread is about! Talking about observations and what we have learned along the way. The only requirement is an open mind when learning.

There are those that have bought their own microscopes and are watching! The Lyme bacteria is seen best with a dark field microscope, but I don't know what requirements must be met for other things, like for studying the immune function.

Why is live blood analysis banned from practice? Even MD's are not allowed to use it for a preliminary diagnosis. I bet more than half of those that tested negative for Lyme antibodies on the Western Blot would be positive if they smeared their blood and looked at it under dark field microscope.

There are many examples of this on YouTube where sick graduates and PhD students used their blood to see if spirochetes were present under the microscope. Thousands of spirochetes, Cyst forms and bio-films can be seem. It just makes you wonder.

IHL said...
maybe IVIG, rituximab, chemo, stem cells, neoplasticism. That's the million dollar question.

My LLMD mentioned stem cells and IVIG to me but I'm not a candidate for the IVIG. I've heard that it has significantly helped chronic Lyme sufferers.

Sorry, I have to lock this thread now. Can't keep posting in this thread.

There is a part 2 and part 3.