aspergillus niger

hey i just get my blood result, and its quite postiiv

aspergillus niger
IGG 124 >50
IGA negativ
IGM 250 >50

somebody has expierence?
thank you
julian

was a blood test i can try to uploud..


[url]https://ibb.co/kX8gr73

Post Edited (palermo) : 2/19/2024 10:05:35 AM (GMT-8)

yep - i am struggling with how one would interpret this kind of result from a mold toxicity illness type perspective

the way i would look at it is - rationally - if we have data to say that the test is usually used for pulmonary infections with aspergillus species - as that's when its usually positive - then its probably best to try to rule that out as a cause of the antibodies - before assuming its evidence of something else - especially is that is something more sinister, less well documented and harder to navigate

the fact that this is an anybody test - means that they are detecting antibodies to the fungal material itself -
( rather than the mold toxins ) but whether this occurs in cases where people are breathing in fragments of mold / spores etc - or only when there is infection and grows of the fungus inside the host - i do not know

i would guess that IgE antibodies ( those associated with allergic reactions ) are possible from inhaled spores in people who are allergic to them - but i dont know if IgG or IgM antibodies form in these cases

as you intimate - i think most credible mold specialists believe mold illness is mainly caused by mold toxins which are typically small molecules from the environment eg in water damaged buildings - and not necessarily mold colonisation ( i know some sources or mold treating practitioners talk about colonisation - but i am not aware of any hard evidence to back this theory up as the general state of play in mold toxin illness patients)

perhaps some of the people who are more knowledgeable with mold have some info on antibody testing ad mold illness

I don’t think it’s “bad” immune status. It shows your immune system responding to something. Either an active threat or existing anti bodies from a past threat. Not inherently good or bad without more data.

Garzie - Dr. Brewer was more into nystatin spray nasally for complex mold patients when they didn’t respond. But I think this was before shoemaker revised marcon treatment to from BEG spray to EDTA+colloidal silver which he now says will address nasal colonization regardless. (Edit no I think he’s still into that) “A controversy surrounds whether patients may harbor mold in their sinuses which then produce mycotoxins in our body. Dr. Shoemaker is adamantly opposed to the idea whereas Dr. Brewer is very convincing of the opposite. “

good input thanks Loski

its new information to me - as its not an area that i have dug into - more just bumped into on the periphery of other topics i have researched more

i cannot speak authoratively on the topic - but what puzzles me a little is; i would have thought it a relatively simple process to stick a endoscope up someone nose and photograph mold colonisation if it exists in a patient

that's the route they go in with the scope in a gastroscopy for instance -
and we know aspergillus forms recognisable well defined fungal colonies in airway infections in the lung - so i would have thought you would see some kind of fungal mass or biofilm if it was present in the nasal passages..

i have seen these kind of images in patients with rare fungal infections - eg the 1 in 100 million type cases where a person gets something ultra rare that normally only grows on some specific plant material.

i am not heavily invested in this earther way - so just carrying on the discussion for the sake of conversation

ref - maybe only a kind of v slight infection vs something like an obvious mass

maybe - but even then, when fungal species grow they form hyphal cells - these are long threads of hair like chains of cells that will grow into a layer of fibrous fungal tissue on a surface. this the hallmark of growth - as apposed to just a few spores or cells settling on a surface and sitting there inactive. therefore colonisation requires growing hyphal threads ( there are yeasts - which are a subset of fungi that can grow by budding as well as by hyphae - but this does not apply to molds like aspergillus)

hyphal threads are actually pretty easy to detect - eg just by swabbing the area - transferring onto a slide and staining with basic microscopy techniques - fungal hyphal cells can easily be distinguished from bacterial cells and other artefacts
you could probably even do it at home - but you could certainly do it with an endoscope to guide the sampling.

also - if there were colonisation - even the lightest growth of fungus on the surface- you would also expect some immune activation locally - so inflammation redness etc - exactly as is seen with candida in the mouth or throat or anywhere else in the body ( it also grows hyphal threads when it colonises )

so again - i just don't see it being that hard to demonstrate - if it indeed did exist in the majority of mold patients

for reference this is what hyphal fungal cells look like in molds like aspergillus
https://ibb.co/2q0mrhw