So you have a normal ileum (where the large and small intestine join). An important distinction that often rules out the majority of Crohn's cases which typically have ileum involvement (always outliers/exceptions but we're talking the bulk of cases, here).
Your large intestine shows erosion (much less in scope than an ulcer) of the normal mucosa surface and obvious reddening (hyperemia) both of which are suggestive of inflammation of any of many possible causes. Often UC patients have those things, but other causes must first be ruled out.
Let's look at the things that sound like an UC:
1.) You have "Visible Crypt Abscess Formation," with the crypts being of particular interest in a UC diagnosis. The intestinal surface has high points and low points, and in medical jargon the low points are known as crypts. Your crypts are clogged with junk that doesn't belong there (usually composed of immune cells like WBCs). This is a common finding with UC patients.
2.) You have "inflammatory cell infiltration" (I'd assume into the shallow, Lamina Propia Layer of tissue, but that is not specified) which means immune cells (typically WBCs/lymphocytes) are attacking something below your protective mucosa barrier. Weaknesses (like the aforementioned erosions) make our defensive mucosa layer weak and permeable, and opportunistic bacteria go into our tissue and start causing troubles, and an immune attack is summoned to confront them.
3.) You have consistent inflammation within the ascending (right side), transverse (horizontal), and descending (left) colon segments, including the rectum. If this is an UC then it would be considered to be a Pancolitis (maximum extent of UC involvement throughout the entire large intestine).
These three things are suggestive of an UC but not definitive.
What's important to UC and NOT mentioned:
1.) None of the above mentions chronic, cell architecture changes. Without that, you could have a one-time infection. With a UC diagnosis you'd expect to see something like "disrupted vascular pattern" (your blood vessel pattern is NOT orderly and predictable as expected, rather is chaotic), crypt branching/dropout, cell wall thickening, and other similar things.
Without chronic architecture changes, I can understand the ambiguity in your diagnosis. Maybe the scope was done earlier in this process, before those architectural changes could occur. Maybe you do NOT have an IBD at all (it coincidentally looks a little like UC). I'm no doctor or pathologist, but I'd say you do not have a storybook, easy to identify as UC case.
Post Edited (iPoop) : 8/23/2018 8:49:48 AM (GMT-6)