Somedude
www.dadamo.com/knowbase/PATHbase/depict.cgi?60"Evidence suggests that patients with ulcerative colitis have elevated antibodies to opposing blood groups. This probably represents the production of blood group like substances from the mucins of the intestinal lining by bacteria in the gut. If so, it may help explain why Sulpha drugs, which work by suppressing the levels of gut bacteria, are generally effective in controlling inflammation. The intestinal microflora are known to be disturbed inflammatory bowel disease.(3) and it has been thought that ulcerative colitis patients were mounting some sort of allergic hypersensitivity to gut bacteria, so depressing the levels of bacteria reduces the immune provocation.
However, it may be specific strains of bacteria capable of degrading the mucin of colitis patients into antigens which mimic an opposing blood type are the true cause of the immune reaction in inflammatory bowel disease. For example, bacteria producing the B antigen in the mucous of a colitis patient who is type A who cause the immune system of the patient to mistake their own colon lining for a bad blood transfusion, and attack it as foreign. This has been reported in the literature, and several strains of bacteria, including species of Proteus vulgaris, a common gut bacteria, are capable of synthesizing a blood type B antigen out of colon mucous called "pseudo-B". "
jid.oxfordjournals.org/content/24/5/428.extractI always love when I find things from like, 1911?!?!? that seem to know more about
things then modern day gis
www.jbc.org/content/9/6/491.full.pdfdo you react to milk?
I do not have time to reformat this but read the study about
the monkeys on page 498
www.jbc.org/content/9/6/491.full.pdfREAD IT
If that doesnt sound like UC, I dunno what does.