Atypical disorder of LES relaxations or non specific motility diosrder

ATYPICAL DISORDERS OF LOS RELAXATION

Patients with atypical disorders of LOS relaxation have one or more manometric features that preclude a diagnosis of classic achalasia, including: (1) some preserved peristalsis, (2) oesophageal contractions with amplitudes >40 mm Hg, and (3) complete LOS relaxation of inadequate duration.20 24-26 These patients have dysphagia, usually for liquids as well as solids, that is not explained by fixed stenoses or inflammation in the oesophagus. The barium swallow often, but not invariably, shows features suggestive of classic achalasia with a dilated oesophagus that terminates in a beak-like narrowing caused by the dysfunctional LOS. Although evidence of inadequate LOS relaxation is found in all cases, the inadequacy may not be apparent by standard manometric analyses. For example, some patients exhibit LOS relaxation that is complete in amplitude but inadequate in duration (that is, the LOS contracts before it is traversed by the peristaltic wave).26 Duration of LOS relaxation is not reported routinely by most motility laboratories. Although the atypical disorders of LOS relaxation can be suspected on the basis of symptoms and manometric features, confirmation of the diagnosis ultimately requires relief of dysphagia by a treatment that decreases resting LOS pressure such as calcium channel blockers, pneumatic dilation, Heller myotomy, or botulinum toxin injection.

It is not clear that identification of the atypical manometric features has any specific clinical or therapeutic implications. Some investigators have proposed that chest pain may be more prominent in patients with a variant of achalasia that is characterised by oesophageal contractions with amplitudes >40 mm Hg (so-called “vigorous achalasia”) whereas others have found that such patients cannot be distinguished clinically from those with classic achalasia.20 One group has reported that injection of botulinum toxin is more effecttive for treating vigorous than classic achalasia30 whereas another group found no such difference in the response to toxin injection.31 Rather than assigning names of dubious significance (for example, vigorous achalasia) to the atypical disorders of LOS relaxation, it seems preferable to use descriptive terms (for example, inadequate LOS relaxation with preserved peristalsis or inadequate LOS relaxation with preserved peristalsis and oesophageal contractions with amplitudes >40 mm Hg, etc.).

I found the above medical explanation on the internet...

According to my results I believe that I fall into this category. So what options I have???? To suffer continually for the rest of my life???
Anyone else with the same manometric findings please?